| 1. | TRPC1, TRPC3, and TRPC6 channels are all involved in cardiac hypertrophy.
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| 2. | The newer Harvard study claimed GDF11 does in fact reverse age-related cardiac hypertrophy.
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| 3. | More recently, Endonuclease G is considered a determinant of cardiac hypertrophy.
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| 4. | However, S100A1-/-did not show the eventual cardiac hypertrophy or chamber dilation in aged mice.
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| 5. | ATGL deficient mice administered pirinixic acid demonstrated reduced cardiac hypertrophy and improved cardiac function.
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| 6. | Decreasing intramitochondrial allows for reversal of cardiac hypertrophy caused in the original cardiac response.
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| 7. | CsA has been shown to decrease cardiac hypertrophy by affecting cardiac myocytes in many ways.
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| 8. | A study of congenital heart defect ( CHD ) investigated the septal defects and cardiac hypertrophy.
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| 9. | Ciclosporin has been used experimentally to treat cardiac hypertrophy ( an increase in cell volume ).
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| 10. | These data suggest that upregulation of talin-1 in cardiac hypertrophy may be detrimental to cardiomyocytes function.
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